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dc.contributor.authorBarber, PAen_NZ
dc.contributor.authorKrishnamurthi, Ren_NZ
dc.contributor.authorParag, Ven_NZ
dc.contributor.authorAnderson, NEen_NZ
dc.contributor.authorRanta, Aen_NZ
dc.contributor.authorKlifoyle, Den_NZ
dc.contributor.authorWong, Een_NZ
dc.contributor.authorGreen, Gen_NZ
dc.contributor.authorArroll, Ben_NZ
dc.contributor.authorBennett, DAen_NZ
dc.contributor.authorWitt, Een_NZ
dc.contributor.authorRush, Een_NZ
dc.contributor.authorSuh, FMen_NZ
dc.contributor.authorTheadom, Aen_NZ
dc.contributor.authorRathnasabapathy, Yen_NZ
dc.contributor.authorTe Ao, Ben_NZ
dc.contributor.authorParmar, Pen_NZ
dc.contributor.authorFeigin, Ven_NZ
dc.contributor.authorfor the ARCOS IV study groupen_NZ
dc.date.accessioned2017-07-27T00:30:51Z
dc.date.available2017-07-27T00:30:51Z
dc.date.copyright2016en_NZ
dc.identifier.citationStroke, STROKEAHA-116.
dc.identifier.urihttp://hdl.handle.net/10292/10698
dc.description.abstractBackground and Purpose—There have been few recent population-based studies reporting the incidence (first ever) and attack rates (incident and recurrent) of transient ischemic attack (TIA). Methods—The fourth Auckland Regional Community Stroke study (ARCOS IV) used multiple overlapping case ascertainment methods to identify all hospitalized and nonhospitalized cases of TIA that occurred in people ≥16 years of age usually resident in Auckland (population ≥16 years of age is 1.12 million), during the 12 months from March 1, 2011. All first-ever and recurrent new TIAs (any new TIA 28 days after the index event) during the study period were recorded. Results—There were 785 people with TIA (402 [51.2%] women, mean [SD] age 71.5 [13.8] years); 614 (78%) of European origin, 84 (11%) Māori/Pacific, and 75 (10%) Asian/Other. The annual incidence of TIA was 40 (95% confidence interval, 36–43), and attack rate was 63 (95% confidence interval, 59–68), per 100 000 people, age standardized to the World Health Organization world population. Approximately two thirds of people were known to be hypertensive or were being treated with blood pressure–lowering agents, half were taking antiplatelet agents and just under half were taking lipid-lowering therapy before the index TIA. Two hundred ten (27%) people were known to have atrial fibrillation at the time of the TIA, of whom only 61 (29%) were taking anticoagulant therapy, suggesting a failure to identify or treat atrial fibrillation. Conclusions—This study describes the burden of TIA in an era of aggressive primary and secondary vascular risk factor management. Education programs for medical practitioners and patients around the identification and management of atrial fibrillation are required.
dc.publisherAmerican Heart Association
dc.relation.urihttp://stroke.ahajournals.org/content/47/9/2183
dc.rightsIf your institution has a policy requiring your manuscript to be deposited in an institutional repository, the AHA CTA grants you those rights. The manuscript should be available in the institutional repository but made publicly accessible no earlier than 6 months after publication.
dc.subjectAnticoagulant; Atrial fibrillation; Cerebrovascular disorders; Hypertension incidence
dc.titleIncidence of Transient Ischemic Attack in Auckland, New Zealand, in 2011 to 2012en_NZ
dc.typeJournal Article
dc.rights.accessrightsOpenAccessen_NZ
dc.identifier.doi10.1161/STROKEAHA.116.014010en_NZ
aut.relation.endpage2188
aut.relation.issue9en_NZ
aut.relation.startpage2183
aut.relation.volume47en_NZ
pubs.elements-id208743
aut.relation.journalStrokeen_NZ


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